Corynoxine triggers cell death via activating PP2A and regulating AKT-mTOR/GSK3β axes in NSCLC - ScienceDirect
High-Mobility Group - an overview
Cells, Free Full-Text
HMGB1 is involved in autophagy inhibition caused by SNCA/α-synuclein overexpression: a process modulated by the natural autophagy inducer corynoxine B. - Abstract - Europe PMC
Chris TAN, Associate Professor, Bachelor of Science, Southern University of Science and Technology, Shenzhen, SUSTech, Department of Chemistry
Calpain inhibition rescues DSS colitis. Hmgb1 fl/fl and Vil-Cre Hmgb1
Juxian SONG, Research Assistant Professor, PhD, Hong Kong Baptist University, Kowloon, School of Chinese Medicine
Chris TAN, Associate Professor, Bachelor of Science, Southern University of Science and Technology, Shenzhen, SUSTech, Department of Chemistry
Corynoxine triggers cell death via activating PP2A and regulating AKT-mTOR/GSK3β axes in NSCLC - ScienceDirect
Jiahong Lu's research works University of Macau, Macau and other places
HMGB1/2 acted as a target for corynoxine B (Cory B) to promote
Frontiers Immunological Significance of HMGB1 Post-Translational Modification and Redox Biology