BKS db Control Mouse Bladder Smooth Muscle Cells

Experimental long-term diabetes mellitus alters the transcriptome and biomechanical properties of the rat urinary bladder

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Urinary Bladder Contraction and Relaxation: Physiology and Pathophysiology

Lifeline® Normal Human Primary Bladder Smooth Muscle Cells (HBSMC), when grown in VascuLife® SMC Smooth Muscle Cell Culture Medium, provide an ideal

Normal Human Bladder Smooth Muscle Cells, Primary

Antagonizing cholecystokinin A receptor in the lung attenuates obesity-induced airway hyperresponsiveness

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Rat Primary Bladder Epithelial Cells

Figure 2 from Central role of the BK channel in urinary bladder smooth muscle physiology and pathophysiology.

Experimental long-term diabetes mellitus alters the transcriptome and biomechanical properties of the rat urinary bladder

Full article: Transcriptional Suppression of CPI-17 Gene Expression in Vascular Smooth Muscle Cells by Tumor Necrosis Factor, Krüppel-Like Factor 4, and Sp1 Is Associated with Lipopolysaccharide-Induced Vascular Hypocontractility, Hypotension, and

A Cellular Reference Resource for the Mouse Urinary Bladder

A Cellular Reference Resource for the Mouse Urinary Bladder

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Proposed physiological roles of MaxiK channel --subunits, depicted as

Spatiotemporal mapping of sensory and motor innervation of the embryonic and postnatal mouse urinary bladder - ScienceDirect