Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect
Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect
C3d-Targeted factor H inhibits tissue complement in disease models and reduces glomerular injury without affecting circulating complement - ScienceDirect
A synthetic protein as efficient multitarget regulator against complement over-activation
Glomerular Disease of Complement Dysregulation (C3 Glomerulopathy, Atypical Hemolytic-Uremic Syndrome)
Recurrent C3 glomerulopathy after kidney transplantation - ScienceDirect
PDF) Complement as a Diagnostic tool in Immunopathology
Complement-mediated kidney diseases - ScienceDirect
Complement dysregulation in glomerulonephritis - ScienceDirect
Complement activation, regulation, and molecular basis for complement‐related diseases
The role of the alternative pathway of complement activation in glomerular diseases